INFLAMMATORY MARKERS IN CHRONIC DIABETIC COMPLICATIONS
INFLAMMATORY MARKERS (Português (Brasil))

Keywords

Diabetes Mellitus, Inflammation, Inflammatory Markers, chronic complications, chronic disease

How to Cite

Thaina De Santos Silva, L. ., & Paula De Sousa Figueiredo, M. . (2023). INFLAMMATORY MARKERS IN CHRONIC DIABETIC COMPLICATIONS: A LITERATURE REVIEW. Health and Society, 3(03), 294–314. https://doi.org/10.51249/hs.v3i03.1415

Abstract

Diabetes Mellitus (DM) is a chronic disease that has been questioned the most because it has become a public health issue worldwide. The state of inflammation may have had an origin in the inflammatory diet, a lack of exercise or other factors associated with Diabetes Mellitus (DM). This work aims to carry out a systematic literature review evaluating the effect of inflammation on chronic complications of Diabetes Mellitus. The study is a bibliographical research, consisting of books and scientific articles. A literature review on the subject will be carried out through scientific academic journals and articles, through databases such as Scielo, Periodicals and Google Scholar, in order to compare the different information found on these platforms, addressing the treatment of Diabetes Mellitus. Articles were searched using the following descriptors: Inflammatory Markers, Diabetes Mellitus, Inflammation, Epidemiology of Diabetes Mellitus. 20 articles were searched, of which 40% addressed the epidemiology of DM, 25% addressed the main inflammatory markers.

https://doi.org/10.51249/hs.v3i03.1415
INFLAMMATORY MARKERS (Português (Brasil))

References

Smeltzer S, Bare B. Histórico e tratamento de pacientes com diabetes mellitus. In: Smeltzer SC, Bare BG. Brunner & Suddarth: tratado de enfermagem médico-cirúrgica. 9th ed. Rio de Janeiro: Guanabara Koogan; 2009. p. 933-83.

Oishi JC. Cinética do desenvolvimento de alterações no perfil inflamatório, função endotelial e cardiovascular na obesidade experimental [tese]. São Carlos: Universidade Federal de São Carlos; 2016.

BATES, M.B., BECHARA, G.H., SEDGWICK, A.D., SIN, Y.M., MACKAY, A. R., WILLOUGHBY, D.A. Effect of a lymphocyte-derived pro-inflammatory factor on carrageenin-pleurisy in the rat. Int. Archs. Allergy appl. Immun., 73:189-190, 1984.

Luft VC, Schmidt MI, Pankow JS, Couper D, Ballantyne CM,YoungJH, Duncan BB Chronic inflammation role in the obesity-diabetes association: a case-cohort study. Diabetol Metab Syndr, 2013.

Duncan BB, Schmidt MI. Chronic activation of the innate immune system may underlie the metabolic syndrome. Rev Paul Med. 2001;119(3):122-7.

Schmidt MI, Duncan BB, Watson RL, Shannett AR, Blancati FL, Heiss G. A metabolic syndrome in whites and african-americans: the atherosclerosis risk in communities (ARIC) baseline study. Diabetes Care. 1996;19(5):414-8.

Grundy SM, Cleeman JI, Daniels SR, Donato KA, Eckel RH, Franklin BA, et al. Diagnosis and management of the metabolic syndrome. An American Heart Association/National Heart, Lung, and Blood Institute (AHA/NHLBI). Scientific statement. Circulation. 2005;112: 2735-52.

International Diabetes Federation. The IDF consensus worldwide definition of the metabolic syndrome. (IDF 2005) - 7p

MATYSZAK, M.K. Inflammation in the Cns: Balance between Immunological Privilege and Immune Responses. Progress in Neurobiology 1998; 56:19-35.

SCHNELL, L.; FEARN, S.; KLASSEN, H.; SCHWAB, M.E.; PERRY, V.H. Acute Inflammatory Responses to Mechanical Lesions in the Cns: Differences betweenBrain and Spinal Cord. European Journal ofNeuroscience 1999; 11 :3648-3658.

GOMES-LEAL, W. Injlamação Aguda, Resposta Glial e Degeneração Axonal em um Modelo de Excitotoxicidade na Medula Espinhal. 20D2. 197f. Tese (Pós-Graduação em Ciências Biológicas -Área de concentração em Neurociências) -Centro de Ciêricias Biológicas, Uni~ersidade Federal do Pará, Belém, 2002.

GOMES-LEAL, W.; CORKILL, D.J.; PICANÇO-DINIZ, C. W. Systematic Analysis of Axonal Damage and Inflammatory Responsem in DifferentWhite MatterTracts ofAcutely Injured Rat Spinal Cord. Brain Research 2005; 1066:57-70.

Pradhan, A. D., Manson, J. E., Rifai, N., Buring, J. E., & Ridker, P. M. (2001). C-reactive protein, interleukin 6, and risk of developing type 2 diabetes mellitus. JAMA, 286(3), 327-334.

Navarro-González, J. F., Mora-Fernández, C., & Muros de Fuentes, M. (2008). Inflammatory molecules and pathways in the pathogenesis of diabetic nephropathy. Nature Reviews Nephrology, 4(6), 327-336.

Hotamisligil, G. S., Shargill, N. S., & Spiegelman, B. M. (1993). Adipose expression of tumor necrosis factor-alpha: direct role in obesity-linked insulin resistance. Science, 259(5091), 87-91.

Pickup, J. C. (2004). Inflammation and activated innate immunity in the pathogenesis of type 2 diabetes. Diabetes Care, 27(3), 813-823.

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This work is licensed under a Creative Commons Attribution 4.0 International License.

Copyright (c) 2023 Larissa Thaina De Santos Silva, Maria Paula De Sousa Figueiredo

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